Speaking of renal failure may be a lot of people will be very afraid, because
we live in the surrounding some families may have lost their lives because of
kidney failure, so we for such diseases are very panic, which should encourage
more people to know more about basic knowledge of renal disease, let's look at
what a reason renal disease caused by?
Cause of renal failure in patients with acute tubular necrosis varied, can be
summarized into two categories:
A renal toxicity to the kidney toxic substances, such as sulfa drugs, carbon
tetrachloride, mercury agents, bismuth, dichloro-sulfamethoxazole
(dichlorphenamide); polymyxin antibiotics, vancomycin, kanamycin Su, gentamicin,
neomycin Pioneer Ⅰ, cephalosporin Ⅱ, neomycin, amphotericin B, and the iodine
contrast agent, methoxy halothane anesthetics; biological toxins such as snake
venom, bee venom, fish, mushroom, cantharidin (Cantharidin), can cause acute
tubular necrosis, under certain conditions.
Second, severe renal ischemia and renal ischemia such as severe trauma,
extensive burns, major surgery, massive blood loss, obstetric hemorrhage, severe
infections, sepsis, dehydration and electrolyte imbalance, particularly with
shock who are easily lead to acute tubular necrosis.
In addition, intravascular hemolysis (eg blackwater fever, primaquine-induced
hemolysis, bean disease, ABO blood transfusion, the oxidation of arsenic
poisoning) released from hemoglobin, as well as muscle mass trauma (such as
crush injuries, muscle inflammation) when myoglobin, by renal excretion, renal
tubular damage may cause acute tubular necrosis. Hereditary nephropathy: as
polycystic kidney disease, Alport syndrome.
Specific pathogenesis of acute tubular necrosis, is not yet fully understood.
It is related to the occurrence of the following:
① extremely reduced glomerular filtration rate (often in 5ml / min or less,
most only 1-2ml / min) generation mechanism may be due to various causes tubular
preceding ischemia or intoxication, renal tubular epithelial cell injury occurs
so that the proximal tubule reabsorption of sodium reduction, so that the
original urine sodium, increase the amount of water. When it flows through the
distal convoluted tubule macula densa, juxtaglomerular stimulation
(juxtaglomerularapparatus) release of renin, angiotensin Ⅱ so that increased
activity in the kidney, causing contraction of glomerular small artery spasm,
leading to glomerular especially in the outer cortical blood flow decreased
glomerular, filtration rate is extremely reduced.
② tubule lumen obstruction after injured necrosis, loss of renal tubular
epithelial cells and inflammatory exudate, blood (muscle) red protein, forming
clumps and tube, blocking the lumen, so that the original dirty urine is
blocked, thus oliguria; on the other hand lumen urine swelling, will increase
pressure on the kidney, the glomerular filtration rate decline further.
③ renal tubular wall rupture, the original urine spill. After the injured
tubules, wall rupture, the original urine tube to tube overflow, thus oliguria;
while renal interstitial edema caused by increased pressure on the kidney, the
glomerular filtration rate.
2015年1月30日星期五
Renal failure can be contagious
In life there are always some people wonder whether renal disease infection,
because it is a very serious kidney disease, and difficult to treat, so there
are many patients with renal failure in life are discriminated against others,
small I do not know why would be so cold, but the fact is that kidney failure is
not contagious.
Renal failure is the development of a variety of chronic kidney disease to kidney function caused by late or part of a pathological state of all the loss. Progression of renal failure can be divided into acute renal failure and chronic renal failure, acute renal failure quickly, usually caused by inadequate blood supply of the kidneys (such as trauma or burns), kidney function due to some factors that cause damage or obstruction by poison injury, acute renal failure caused by generation. The main reason for the long-term chronic renal failure kidney disease, along with the time and the disease, decreased kidney function, resulting in renal failure.
Acute renal failure, the disease is usually because our kidneys because of lack of blood supply, or by some viruses, cause kidney injury, which is generally faster onset of acute renal dysfunction can cause acute renal failure generation. Chronic renal failure, this is mainly due to the long-term so that their kidneys hurt, renal lesion, and over time, resulting in our slowly lost kidney function, leading to chronic renal failure occur.
Here you first need to say is that for the average kidney disease are basically not contagious, and for renal failure is the same, the situation does not appear infection, renal failure, the cause of this disease is more complex, the main the reason is that the human glomerular serious damage caused.
Renal failure is a part of urology disease, so this is a case of contagious disease does not occur, the family and the people around you do not need to worry about, for renal failure can be cured, so patients have found themselves in this after the disease, be sure to keep a good attitude, aggressive treatment,
Renal failure is our human kidney function has all or part of the loss of function caused by a disease, renal failure is classified into acute and chronic failure, generally more rapid onset of acute failure, symptoms are obvious, chronic failure is also more serious and need attention.
Each with contagious diseases are not so dear friends, do not be too alarmist, need to understand the fact that after the act, otherwise, would have been very easy for patients sad cause much harm ah. Renal failure is not contagious disease, we must remember clearly.
Renal failure is the development of a variety of chronic kidney disease to kidney function caused by late or part of a pathological state of all the loss. Progression of renal failure can be divided into acute renal failure and chronic renal failure, acute renal failure quickly, usually caused by inadequate blood supply of the kidneys (such as trauma or burns), kidney function due to some factors that cause damage or obstruction by poison injury, acute renal failure caused by generation. The main reason for the long-term chronic renal failure kidney disease, along with the time and the disease, decreased kidney function, resulting in renal failure.
Acute renal failure, the disease is usually because our kidneys because of lack of blood supply, or by some viruses, cause kidney injury, which is generally faster onset of acute renal dysfunction can cause acute renal failure generation. Chronic renal failure, this is mainly due to the long-term so that their kidneys hurt, renal lesion, and over time, resulting in our slowly lost kidney function, leading to chronic renal failure occur.
Here you first need to say is that for the average kidney disease are basically not contagious, and for renal failure is the same, the situation does not appear infection, renal failure, the cause of this disease is more complex, the main the reason is that the human glomerular serious damage caused.
Renal failure is a part of urology disease, so this is a case of contagious disease does not occur, the family and the people around you do not need to worry about, for renal failure can be cured, so patients have found themselves in this after the disease, be sure to keep a good attitude, aggressive treatment,
Renal failure is our human kidney function has all or part of the loss of function caused by a disease, renal failure is classified into acute and chronic failure, generally more rapid onset of acute failure, symptoms are obvious, chronic failure is also more serious and need attention.
Each with contagious diseases are not so dear friends, do not be too alarmist, need to understand the fact that after the act, otherwise, would have been very easy for patients sad cause much harm ah. Renal failure is not contagious disease, we must remember clearly.
What are some natural remedies for kidney stones
In the incidence of kidney stones among the crowd, male patients than female
or more, for a large number of men drinking, if the case of kidney stones exist,
the need for early drinking, and also as soon as possible to a regular hospital
treatment professional nephropathy. So what kidney stones Naturopathy is it?
Let's hear how the experts say it!
1, if the local water is hard water calcium or high water, the stone is definitely high incidence. If the patient or the patient normally have the habit of drinking tea are not a lot of drinking habits, but also prone to stones.
2, stones occur, there may be a relationship with the patient's other diseases, the opportunity if hyperparathyroidism in patients with kidney stones occur is high.
3, stones occur also career-related, such as steel workers, normally sweating a lot, prone to stones.
4, the occurrence of kidney stones, and diet are closely related, such as spinach, red bayberry, tomatoes, cocoa, chocolate, pepper, potatoes, peppers, tofu and so easy to promote the deposition of impurities in the urine of food.
5, if not a simple movement of calcium, but also easy to generate stones. In fact, the largest movement of calcium absorption, bone formation because then you need exercise, so athletes are the most stout bones, exercise can help to precipitate calcium bones, but do not exercise, it will precipitate calcium from the bones into the blood inside, into the urine fluid go in. So simply do not exercise will increase calcium stones occur.
1, if the local water is hard water calcium or high water, the stone is definitely high incidence. If the patient or the patient normally have the habit of drinking tea are not a lot of drinking habits, but also prone to stones.
2, stones occur, there may be a relationship with the patient's other diseases, the opportunity if hyperparathyroidism in patients with kidney stones occur is high.
3, stones occur also career-related, such as steel workers, normally sweating a lot, prone to stones.
4, the occurrence of kidney stones, and diet are closely related, such as spinach, red bayberry, tomatoes, cocoa, chocolate, pepper, potatoes, peppers, tofu and so easy to promote the deposition of impurities in the urine of food.
5, if not a simple movement of calcium, but also easy to generate stones. In fact, the largest movement of calcium absorption, bone formation because then you need exercise, so athletes are the most stout bones, exercise can help to precipitate calcium bones, but do not exercise, it will precipitate calcium from the bones into the blood inside, into the urine fluid go in. So simply do not exercise will increase calcium stones occur.
2015年1月29日星期四
What is the harm of chronic nephritis
What is the harm of chronic nephritis? According to our survey shows related
to women are very susceptible to chronic nephritis disease, especially young
women friends, so what women suffer from chronic nephritis bring us harm is it,
let's work together to understand it;
Women with chronic nephritis What harm?
Experts, suffering from chronic nephritis is a very long time, this disease is very easy to attack, and now there is no better way in the treatment method, which makes a lot of patients prone to irritability, despair, and even low self-esteem emotional, physical and mental health that will directly harm the patient, the impact of the disease. So harmonious family life, caring and considerate husband and wife are very important, the husband should help his wife the right to treat the disease, maintaining optimism, the message to prevent infection, and rest patterns of life, sex life sparingly, avoid over exertion, thus facilitating stabilization and rehabilitation of chronic nephritis. Chronic nephritis treatment more difficult, and if not treated properly or not timely, there might develop into uremia, endanger life. If women had chronic nephritis, which is more serious, chronic nephritis patients most likely to lead to women unable to have children.
Chronic nephritis associated with chronic renal failure who should not reproduce. Women after pregnancy, renal failure, kidney might be overwhelmed renal dysfunction, resulting in uremia, life-threatening.
What is the harm of chronic nephritis? The experts on women's presented to us by the harm suffered chronic nephritis, female friends should have a better understanding of it, thus requiring everyone with medical treatment in their daily lives and actively care.
If you have any questions you can consult Shijiazhuang kidney disease hospital.
Women with chronic nephritis What harm?
Experts, suffering from chronic nephritis is a very long time, this disease is very easy to attack, and now there is no better way in the treatment method, which makes a lot of patients prone to irritability, despair, and even low self-esteem emotional, physical and mental health that will directly harm the patient, the impact of the disease. So harmonious family life, caring and considerate husband and wife are very important, the husband should help his wife the right to treat the disease, maintaining optimism, the message to prevent infection, and rest patterns of life, sex life sparingly, avoid over exertion, thus facilitating stabilization and rehabilitation of chronic nephritis. Chronic nephritis treatment more difficult, and if not treated properly or not timely, there might develop into uremia, endanger life. If women had chronic nephritis, which is more serious, chronic nephritis patients most likely to lead to women unable to have children.
Chronic nephritis associated with chronic renal failure who should not reproduce. Women after pregnancy, renal failure, kidney might be overwhelmed renal dysfunction, resulting in uremia, life-threatening.
What is the harm of chronic nephritis? The experts on women's presented to us by the harm suffered chronic nephritis, female friends should have a better understanding of it, thus requiring everyone with medical treatment in their daily lives and actively care.
If you have any questions you can consult Shijiazhuang kidney disease hospital.
Prevention of lupus kidney disease
Lupus kidney disease prevention. For lupus disease do you know how much? Many
of the patients in understanding this disease is relatively large relatively
small, even if there is rarely a real insight into the patients with this
disease, kidney disease Given this is a serious threat to patient safety one
disease, kidney disease treatment experts believe prevention of this disease or
have a need to look at it!
What lupus kidney disease prevention methods have it?
First, pay attention to the physical elements
The incidence of systemic lupus erythematosus relatives up to 5% to 12%, the incidence of monozygotic twins up to 69% of lupus patients with relatives in other autoimmune diseases such as rheumatoid, dermatomyositis, scleroderma, psoriasis and other dry disease and morbidity too high, black and Asian high incidence of blue-eyed children heirs, which are influenced by genetic factors prompted by lupus. The woman multiple, low testosterone level of patients, endocrine factors are clarified, especially estrogen in the pathogenesis of lupus patient level and lupus nephritis related. Thus, with regard to the foundation has its own immune disease lupus patients, including relatives of those who have autoimmune disease, should be vigilant, once the disease should think itself immune disease, once suffering from autoimmune disease, treatment should be active, prevent the onset of lupus nephritis resulting in the formation of renal damage.
Second, the active virus infection treatment
In recent years, experimental studies have found that virus infection may be related to the onset of systemic lupus erythematosus related. Therefore, we should actively treat a variety of viral infections. Particularly with regard to the upper respiratory tract viral infections such "minor ailments", must not casually. As the "wind is riddled with long," many are ill with colds for the beginning of the incidence and thus endanger the population.
Third, pay attention to drug toxicity
Associated with lupus drug hydralazine, procainamide, isoniazid, methyldopa, chlorprothixene it triazine and quinidine, especially once the two is more common. Perhaps with drugs hydrazine, amine, thiol groups concerned. Thus on lupus patients have lupus foundation of genetics, these drugs should be noted that perhaps the onset of lupus patients toxicity.
Fourth, to prevent sun exposure
Ultraviolet radiation aggravate the condition is more common in lupus nephritis. Because ultraviolet dna can be converted to thymine dimers, leaving antigen enhanced to promote the onset of systemic lupus erythematosus. Thus, patients with lupus nephritis should be prevented in the usual day in the fierce sunlight prolonged exposure to excessive ultraviolet shine reduce kidney damage formation.
What lupus kidney disease prevention methods have it?
First, pay attention to the physical elements
The incidence of systemic lupus erythematosus relatives up to 5% to 12%, the incidence of monozygotic twins up to 69% of lupus patients with relatives in other autoimmune diseases such as rheumatoid, dermatomyositis, scleroderma, psoriasis and other dry disease and morbidity too high, black and Asian high incidence of blue-eyed children heirs, which are influenced by genetic factors prompted by lupus. The woman multiple, low testosterone level of patients, endocrine factors are clarified, especially estrogen in the pathogenesis of lupus patient level and lupus nephritis related. Thus, with regard to the foundation has its own immune disease lupus patients, including relatives of those who have autoimmune disease, should be vigilant, once the disease should think itself immune disease, once suffering from autoimmune disease, treatment should be active, prevent the onset of lupus nephritis resulting in the formation of renal damage.
Second, the active virus infection treatment
In recent years, experimental studies have found that virus infection may be related to the onset of systemic lupus erythematosus related. Therefore, we should actively treat a variety of viral infections. Particularly with regard to the upper respiratory tract viral infections such "minor ailments", must not casually. As the "wind is riddled with long," many are ill with colds for the beginning of the incidence and thus endanger the population.
Third, pay attention to drug toxicity
Associated with lupus drug hydralazine, procainamide, isoniazid, methyldopa, chlorprothixene it triazine and quinidine, especially once the two is more common. Perhaps with drugs hydrazine, amine, thiol groups concerned. Thus on lupus patients have lupus foundation of genetics, these drugs should be noted that perhaps the onset of lupus patients toxicity.
Fourth, to prevent sun exposure
Ultraviolet radiation aggravate the condition is more common in lupus nephritis. Because ultraviolet dna can be converted to thymine dimers, leaving antigen enhanced to promote the onset of systemic lupus erythematosus. Thus, patients with lupus nephritis should be prevented in the usual day in the fierce sunlight prolonged exposure to excessive ultraviolet shine reduce kidney damage formation.
Nephrotic syndrome both What are the performance exist
The existence of nephrotic syndrome of disease of there will be what kind of
the symptoms of it? Now more and and more friends positive suffering from hurt
nephrotic syndrome of the disease, everyone is also the more and more of concern
disease, but for the knowledge understanding of of disease of of or too little.
Therefore, the Xiao Bian today prepared for you are prophase manifestation About
nephrotic syndrome of the disease.
1, hyperlipidemia
Hyperlipidemia with the urine, lipids, fat body low protein hyperlipidemia patient-related of urine birefringence, may be the the epithelial cells or fat body tube-containing cholesterol.
2, a large number of proteinuria
The main component in urine protein in, further contain other plasma protein. Every day, more than 3 the patient's proteinuria. 5 g, of fat urine. Glomerular filtration rate decreased, urinary protein decline; increase severe low protein hyperlipidemia, urinary protein the amount of of, high protein diet will increase the urinary protein excretion.
3, low protein hyperlipidemia
Low protein hyperlipidemia is refers to the has less than 30 g serum albumin in patients with per liter of of, compensate when patients liver synthesis of albumin of the role of is not sufficient to make up for the of urinary the loss of protein amount of, will appear low protein hyperlipidemia.
4, edema
Clinical signs nephrotic syndrome? The most obvious symptom is gradual of systemic sex edema in patients with, the beginning of early morning eyelid, facial, ankle joint edema, With the gradual development edema disease of, will affect the the body, and the emergence of ascites, pleural effusion, pericardium effusion, mediastinal effusion, scrotum or labia edema, also can appear pulmonary edema.
1, hyperlipidemia
Hyperlipidemia with the urine, lipids, fat body low protein hyperlipidemia patient-related of urine birefringence, may be the the epithelial cells or fat body tube-containing cholesterol.
2, a large number of proteinuria
The main component in urine protein in, further contain other plasma protein. Every day, more than 3 the patient's proteinuria. 5 g, of fat urine. Glomerular filtration rate decreased, urinary protein decline; increase severe low protein hyperlipidemia, urinary protein the amount of of, high protein diet will increase the urinary protein excretion.
3, low protein hyperlipidemia
Low protein hyperlipidemia is refers to the has less than 30 g serum albumin in patients with per liter of of, compensate when patients liver synthesis of albumin of the role of is not sufficient to make up for the of urinary the loss of protein amount of, will appear low protein hyperlipidemia.
4, edema
Clinical signs nephrotic syndrome? The most obvious symptom is gradual of systemic sex edema in patients with, the beginning of early morning eyelid, facial, ankle joint edema, With the gradual development edema disease of, will affect the the body, and the emergence of ascites, pleural effusion, pericardium effusion, mediastinal effusion, scrotum or labia edema, also can appear pulmonary edema.
Symptoms of diabetic nephropathy and staging
Symptoms of diabetic nephropathy and staging. Is very much the type of kidney
disease, and for that matter, most of the friends can say a few years, for
example, the more common kidney uremia, hydronephrosis, etc., but I do not know
does not mean that the disease exist.
What are the symptoms of diabetic nephropathy and staging it?
Reflect
1, no proteinuria early clinical proteinuria in diabetic nephropathy, only by radioimmunoassay talent detect microalbuminuria. Pre-diabetic nephropathy specific clinical manifestation of proteinuria, proteinuria gradually unfold from intermittent to continue resistance.
2, pre-diabetic nephropathy clinical edema usually no edema, a small number of patients before lowering plasma protein, may have mild edema. If a lot of proteinuria, low plasma protein, increased edema, mostly carried out to late disease expression.
3, high blood pressure in type 1 diabetic patients without nephropathy prevalence of hypertension than normal does not add, type 2 diabetes and hypertension are more, but if the time share presentation hypertension also increased proteinuria in kidney disease when syndrome patients with hypertension, this mostly moderate hypertension, a small number of severe.
4, there is a big difference with renal failure and diabetic nephropathy launch speed. Some patients with mild proteinuria may continue for many years, but with normal renal function, some patients with proteinuria few quick launch presented nephrotic syndrome, renal function progressively worse, eventually showing uremia.
5, anemia in patients with significant azotemia, may have mild anemia.
6, other organ complications such as cardiovascular disease embody heart failure, myocardial infarction. Neuropathy, such as peripheral neuropathy. When presented with neurogenic bladder may involve autonomic nervous. Retinopathy, diabetic nephropathy almost 100% when severe diabetic retinopathy, but there are severe retinopathy significantly divergent set of kidney disease. When carried out with diabetic nephropathy, retinopathy often accelerate deterioration.
Staging
Ⅰ period: increase in glomerular filtration rate and renal volume increases as features. This precancerous lesions and high blood sugar levels together, but reversible, by insulin treatment can be recovered, but divergent set to completely recover to normal.
Ⅱ period: The period of normal urinary albumin excretion rate, but has shown signs of glomerular structural changes. This issue of urinary albumin excretion rate (UAE) normal (<20μg / min or <30mg / 24h), after the group increased after exercise UAE rest can recover. The structure of the glomerular changes have been presented, glomerular basement membrane (GBM) thickening and mesangial matrix spike, GFR much higher than normal blood sugar levels with a common, GFR> 150mL / min in patients with glycated hemoglobin often > 9.5%. GFR> 150mL / min and the UAE> patient 30μg / min easier to conduct future clinical diabetic nephropathy. Diabetic kidney damage Ⅰ, Ⅱ patients blood pressure were normal. Ⅰ, GFR Ⅱ patients increased, UAE normal, therefore can not be called the second phase of diabetic nephropathy.
Stage Ⅲ: also known as pre-diabetic. Urinary albumin excretion rate 20--200μg / min, slightly elevated blood pressure, glomerular Kuang fees began to appear.
Stage Ⅳ: clinical diabetic nephropathy or overt diabetic nephropathy. This period was characterized by a lot of albuminuria (greater than 3.5 grams per day), edema and hypertension. Diabetic nephropathy is more severe edema, diuretics poor response.
Ⅴ stage: the end of advanced renal failure. Once the presentation of diabetes continues to carry out clinical proteinuria in diabetic nephropathy, glomerular basement membrane as a result of extensive thickening of the glomerular capillary cavity narrow and more glomeruli Kuang fees were reduced kidney filtration function , resulting in renal failure
About Diabetic nephropathy staging problem if you have any questions you can consult a online doctor Shijiazhuang kidney disease hospital, we can give you help.
What are the symptoms of diabetic nephropathy and staging it?
Reflect
1, no proteinuria early clinical proteinuria in diabetic nephropathy, only by radioimmunoassay talent detect microalbuminuria. Pre-diabetic nephropathy specific clinical manifestation of proteinuria, proteinuria gradually unfold from intermittent to continue resistance.
2, pre-diabetic nephropathy clinical edema usually no edema, a small number of patients before lowering plasma protein, may have mild edema. If a lot of proteinuria, low plasma protein, increased edema, mostly carried out to late disease expression.
3, high blood pressure in type 1 diabetic patients without nephropathy prevalence of hypertension than normal does not add, type 2 diabetes and hypertension are more, but if the time share presentation hypertension also increased proteinuria in kidney disease when syndrome patients with hypertension, this mostly moderate hypertension, a small number of severe.
4, there is a big difference with renal failure and diabetic nephropathy launch speed. Some patients with mild proteinuria may continue for many years, but with normal renal function, some patients with proteinuria few quick launch presented nephrotic syndrome, renal function progressively worse, eventually showing uremia.
5, anemia in patients with significant azotemia, may have mild anemia.
6, other organ complications such as cardiovascular disease embody heart failure, myocardial infarction. Neuropathy, such as peripheral neuropathy. When presented with neurogenic bladder may involve autonomic nervous. Retinopathy, diabetic nephropathy almost 100% when severe diabetic retinopathy, but there are severe retinopathy significantly divergent set of kidney disease. When carried out with diabetic nephropathy, retinopathy often accelerate deterioration.
Staging
Ⅰ period: increase in glomerular filtration rate and renal volume increases as features. This precancerous lesions and high blood sugar levels together, but reversible, by insulin treatment can be recovered, but divergent set to completely recover to normal.
Ⅱ period: The period of normal urinary albumin excretion rate, but has shown signs of glomerular structural changes. This issue of urinary albumin excretion rate (UAE) normal (<20μg / min or <30mg / 24h), after the group increased after exercise UAE rest can recover. The structure of the glomerular changes have been presented, glomerular basement membrane (GBM) thickening and mesangial matrix spike, GFR much higher than normal blood sugar levels with a common, GFR> 150mL / min in patients with glycated hemoglobin often > 9.5%. GFR> 150mL / min and the UAE> patient 30μg / min easier to conduct future clinical diabetic nephropathy. Diabetic kidney damage Ⅰ, Ⅱ patients blood pressure were normal. Ⅰ, GFR Ⅱ patients increased, UAE normal, therefore can not be called the second phase of diabetic nephropathy.
Stage Ⅲ: also known as pre-diabetic. Urinary albumin excretion rate 20--200μg / min, slightly elevated blood pressure, glomerular Kuang fees began to appear.
Stage Ⅳ: clinical diabetic nephropathy or overt diabetic nephropathy. This period was characterized by a lot of albuminuria (greater than 3.5 grams per day), edema and hypertension. Diabetic nephropathy is more severe edema, diuretics poor response.
Ⅴ stage: the end of advanced renal failure. Once the presentation of diabetes continues to carry out clinical proteinuria in diabetic nephropathy, glomerular basement membrane as a result of extensive thickening of the glomerular capillary cavity narrow and more glomeruli Kuang fees were reduced kidney filtration function , resulting in renal failure
About Diabetic nephropathy staging problem if you have any questions you can consult a online doctor Shijiazhuang kidney disease hospital, we can give you help.
2015年1月26日星期一
24-hour urinary protein excretion what use is it
24-hour urinary protein excretion what use is it? In daily life, many people
will appear in the examination when the phenomenon of high urinary protein, but
do not know how to do that when the time with normal renal function, proteinuria
in the normal range, sometimes there will be a high urine protein phenomenon,
following on from a kidney specialist for everyone for a detailed answer to this
question!
24-hour urinary protein excretion what use is it?
Normal daily urinary excretion of protein is generally 40 to 80 mg, the upper limit is 150 mg, called physiological proteinuria, which is the normal 24-hour urine protein. Due to less conventional laboratory testing was negative. And more than 150 mg / day, this is an unusual phenomenon that urinary protein.
Under normal circumstances, the body in strenuous exercise, heavy labor, emotional, cold, heat, and at the time of stress, urinary protein excretion may increase, called a transient urinary protein, in a few hours or a few days later can be restored to normal. Also patients with kidney disease may also occur in urine protein, called pathological urinary protein, which is more than 24 hours of normal urinary protein, to timely treatment.
Inspection methods urine protein checks and urine protein checks are often taken to check the method of urinary protein qualitative and quantitative examination of two checks. Qualitative examination is placed in the urine drug test or test strips, according to their degree of discoloration of urine to check for protein, quantitative examination is investigating the amount of protein in the urine contained copies a day, from these tests to understand kidney function Are there obstacles.
24-hour urinary protein excretion what use is it? Above is related presentations on 24 hours a day of normal urine protein, hoping to be helpful to everyone, to remind you, if once the examination revealed abnormal, should be treated promptly to avoid the body and life to bring greater harm.
Viber/WhatsApp:+8615176855086
Skype:live:kidneyservice
Email: kidneyservice@hotmail.com
24-hour urinary protein excretion what use is it?
Normal daily urinary excretion of protein is generally 40 to 80 mg, the upper limit is 150 mg, called physiological proteinuria, which is the normal 24-hour urine protein. Due to less conventional laboratory testing was negative. And more than 150 mg / day, this is an unusual phenomenon that urinary protein.
Under normal circumstances, the body in strenuous exercise, heavy labor, emotional, cold, heat, and at the time of stress, urinary protein excretion may increase, called a transient urinary protein, in a few hours or a few days later can be restored to normal. Also patients with kidney disease may also occur in urine protein, called pathological urinary protein, which is more than 24 hours of normal urinary protein, to timely treatment.
Inspection methods urine protein checks and urine protein checks are often taken to check the method of urinary protein qualitative and quantitative examination of two checks. Qualitative examination is placed in the urine drug test or test strips, according to their degree of discoloration of urine to check for protein, quantitative examination is investigating the amount of protein in the urine contained copies a day, from these tests to understand kidney function Are there obstacles.
24-hour urinary protein excretion what use is it? Above is related presentations on 24 hours a day of normal urine protein, hoping to be helpful to everyone, to remind you, if once the examination revealed abnormal, should be treated promptly to avoid the body and life to bring greater harm.
Viber/WhatsApp:+8615176855086
Skype:live:kidneyservice
Email: kidneyservice@hotmail.com
It appears serious 2+ proteinuria
2+ proteinuria appears serious? People appear 2+ proteinuria are more
confused, do not appear to know how it was 2+ proteinuria. In fact, patients
with kidney disease should no stranger to this. In order to allow everyone a
good understanding of 2+ proteinuria, urinary protein 2+ serious? Following on
from the kidney specialists for you on the next 2+ proteinuria.
2+ proteinuria appears serious?
First, let's take a look at the urine protein, urine protein after heating by acidifying the urine turbid and detected proteins. The normal range for 24-hour urine protein ≦ 0.15g, routine laboratory urine protein test is negative. Such as the detection of proteinuria> 150 mg / day, hour urine protein that is positive, indicating that the amount of protein in urine increased significantly discharged from the body, are abnormal urinary protein. Urinary protein remains positive, often on behalf of kidney lesions occurred. Kidney and urinary protein 2+ represents a problem.
In addition, experts say testing, 24-hour urinary protein excretion of normal reference value of 10 to 150 mg. If 150 to 500 milligrams, for microalbuminuria,> 500 mg clinical proteinuria. Tip microalbuminuria in early diabetic nephropathy, need long-term control of blood glucose, to reverse or delay kidney disease and retinopathy progression of some significance.
Normal urine contained no or only trace amounts of protein, test results were negative (-), when urine protein content up to a certain amount is to check the result is positive (+). 2+ proteinuria appears very serious? Urinary protein 2 + (++, two plus) is doing a result 24 hours urine protein detection. Also can be expressed as two plus proteinuria, urinary protein +2, several statements is a meaning. Urinary protein mean plus two 1L urine contains protein 1.0 ~ 2.0g, namely 1.0 ~ 2.0g / L.
2+ proteinuria appears serious? These are the experts to explain 2+ proteinuria on! I hope you can understand that if there is anything you do not understand where you can click on the online experts for consultation, the experts will make a detailed answer to your problem!
anyquestions you can ask Shijiazhuang Kidney Disease Hospital doctors. They will give you the best free advices.
2+ proteinuria appears serious?
First, let's take a look at the urine protein, urine protein after heating by acidifying the urine turbid and detected proteins. The normal range for 24-hour urine protein ≦ 0.15g, routine laboratory urine protein test is negative. Such as the detection of proteinuria> 150 mg / day, hour urine protein that is positive, indicating that the amount of protein in urine increased significantly discharged from the body, are abnormal urinary protein. Urinary protein remains positive, often on behalf of kidney lesions occurred. Kidney and urinary protein 2+ represents a problem.
In addition, experts say testing, 24-hour urinary protein excretion of normal reference value of 10 to 150 mg. If 150 to 500 milligrams, for microalbuminuria,> 500 mg clinical proteinuria. Tip microalbuminuria in early diabetic nephropathy, need long-term control of blood glucose, to reverse or delay kidney disease and retinopathy progression of some significance.
Normal urine contained no or only trace amounts of protein, test results were negative (-), when urine protein content up to a certain amount is to check the result is positive (+). 2+ proteinuria appears very serious? Urinary protein 2 + (++, two plus) is doing a result 24 hours urine protein detection. Also can be expressed as two plus proteinuria, urinary protein +2, several statements is a meaning. Urinary protein mean plus two 1L urine contains protein 1.0 ~ 2.0g, namely 1.0 ~ 2.0g / L.
2+ proteinuria appears serious? These are the experts to explain 2+ proteinuria on! I hope you can understand that if there is anything you do not understand where you can click on the online experts for consultation, the experts will make a detailed answer to your problem!
anyquestions you can ask Shijiazhuang Kidney Disease Hospital doctors. They will give you the best free advices.
Nephrotic syndrome patients can live how long it
You know nephrotic syndrome can be much longer do? Members and friends of
patients, with an optimistic and positive attitude is critical for the cure of
diseases. Nephrotic syndrome patients had friends, do not be afraid to change
their attitude, positive and optimistic treatment.
You know nephrotic syndrome can be much longer do?
Patients and their families always worried nephrotic syndrome can be much longer question whether the symptoms disappear after a relapse, which is understandable. This problem is more complicated to explain.
?? How much longer can the treatment of nephrotic syndrome, experts pointed out that, in general, nephrotic syndrome, the treatment should follow the following principles:
1. Diuretics: usually 7-10 days after the medication that will attend the urine, unnecessary use of diuretics. Nephrotic syndrome patients with severe edema can use diuretics, commonly used diuretics have furosemide, administered intravenously, it is best to lose the low molecular weight dextran oil spin, often have a good diuretic effect.
2. Hormone Therapy: The most commonly used drug is prednisone nephrotic syndrome patients, depending on the type of disease, children respond to prednisone, etc., were using a short course of two months, in the course of 4-6 months and 9-12 months long course. Simple short course for newly diagnosed kidney disease, and in the long course of treatment for complex, multi-simple repeated kidney disease or kidney disease nephritis.
3. immunosuppressive drugs: patients with nephrotic syndrome, the use of hormones or treatment side effects too bad, you can use immunosuppressive therapy, the most commonly used drugs are cyclic amines, sulfur bird glanced Yin, ring full neomycin A, vincristine TWP so on.
However, nephrotic syndrome can be much longer? Because of the enormous hormonal treatment for nephrotic syndrome side effects, many patients choose TCM treatment. However, clinical findings, although the side effects of traditional Chinese medicine treatment, but treatment for nephrotic syndrome, relying solely on traditional Chinese medicine is not enough.
Because the traditional Western medicine treatment of nephrotic syndrome, palliative, if it does not choose a kidney transplant patients will inevitably dying. And I'm Home "machine because of intervention" medicine kidney area under the theoretical guidance ion import this machine due to the intervention of TCM therapy nephritis significant effect, in combination therapy, both for primary disease, but also the treatment of pathological changes, multi-level, multi-faceted adjust the overall homeostasis. When applied in medicine, take long in Western medicine, complement each other, weaknesses, to be simple, to minimize the side effects of medication, shorten treatment, improve efficacy, reduce the financial burden on patients.
You know nephrotic syndrome can be much longer do? So we must believe that, just find the right treatment, together with aspects of daily necessities of life care, the success of the treatment will go on! Nephrotic syndrome can be cured, I believe science, to create the miracle of life!
You know nephrotic syndrome can be much longer do?
Patients and their families always worried nephrotic syndrome can be much longer question whether the symptoms disappear after a relapse, which is understandable. This problem is more complicated to explain.
?? How much longer can the treatment of nephrotic syndrome, experts pointed out that, in general, nephrotic syndrome, the treatment should follow the following principles:
1. Diuretics: usually 7-10 days after the medication that will attend the urine, unnecessary use of diuretics. Nephrotic syndrome patients with severe edema can use diuretics, commonly used diuretics have furosemide, administered intravenously, it is best to lose the low molecular weight dextran oil spin, often have a good diuretic effect.
2. Hormone Therapy: The most commonly used drug is prednisone nephrotic syndrome patients, depending on the type of disease, children respond to prednisone, etc., were using a short course of two months, in the course of 4-6 months and 9-12 months long course. Simple short course for newly diagnosed kidney disease, and in the long course of treatment for complex, multi-simple repeated kidney disease or kidney disease nephritis.
3. immunosuppressive drugs: patients with nephrotic syndrome, the use of hormones or treatment side effects too bad, you can use immunosuppressive therapy, the most commonly used drugs are cyclic amines, sulfur bird glanced Yin, ring full neomycin A, vincristine TWP so on.
However, nephrotic syndrome can be much longer? Because of the enormous hormonal treatment for nephrotic syndrome side effects, many patients choose TCM treatment. However, clinical findings, although the side effects of traditional Chinese medicine treatment, but treatment for nephrotic syndrome, relying solely on traditional Chinese medicine is not enough.
Because the traditional Western medicine treatment of nephrotic syndrome, palliative, if it does not choose a kidney transplant patients will inevitably dying. And I'm Home "machine because of intervention" medicine kidney area under the theoretical guidance ion import this machine due to the intervention of TCM therapy nephritis significant effect, in combination therapy, both for primary disease, but also the treatment of pathological changes, multi-level, multi-faceted adjust the overall homeostasis. When applied in medicine, take long in Western medicine, complement each other, weaknesses, to be simple, to minimize the side effects of medication, shorten treatment, improve efficacy, reduce the financial burden on patients.
You know nephrotic syndrome can be much longer do? So we must believe that, just find the right treatment, together with aspects of daily necessities of life care, the success of the treatment will go on! Nephrotic syndrome can be cured, I believe science, to create the miracle of life!
2015年1月25日星期日
IgA nephropathy treatment should be how to prevent?
Primary iga nephropathy should be how to prevent?
Prevent colds and overworked, caution nephrotoxic drugs. Should be treated with antibiotics if the infection. There are chronic recurrent tonsillitis, could of tonsillectomy.
Primary iga nephropathy should be treated?
First, the general treatment
Prevent colds and overworked, caution nephrotoxic drugs. Should be treated with antibiotics if the infection. There are chronic recurrent tonsillitis, possible tonsillectomy.
Second, drug treatment
(A) significant hematuria were available Tripterygium glycosides, dipyridamole, acei oral therapy and a lot of vitamin c.
(B) who was treating nephrotic syndrome nephrotic syndrome, see chapter.
(Three) and chronic glomerulonephritis same person, according to the treatment of chronic glomerulonephritis.
(D) the pathology associated with crescent formation and deposition of the main iga who can strengthen the plasma exchange therapy and drug therapy (see Rapidly progressive glomerulonephritis treatment).
Third, blood purification therapy
When associated with acute renal failure or chronic renal failure have been formed by the need for hemodialysis or peritoneal dialysis.
KIDNEYSERVICE@HOTMAIL.COM
Prevent colds and overworked, caution nephrotoxic drugs. Should be treated with antibiotics if the infection. There are chronic recurrent tonsillitis, could of tonsillectomy.
Primary iga nephropathy should be treated?
First, the general treatment
Prevent colds and overworked, caution nephrotoxic drugs. Should be treated with antibiotics if the infection. There are chronic recurrent tonsillitis, possible tonsillectomy.
Second, drug treatment
(A) significant hematuria were available Tripterygium glycosides, dipyridamole, acei oral therapy and a lot of vitamin c.
(B) who was treating nephrotic syndrome nephrotic syndrome, see chapter.
(Three) and chronic glomerulonephritis same person, according to the treatment of chronic glomerulonephritis.
(D) the pathology associated with crescent formation and deposition of the main iga who can strengthen the plasma exchange therapy and drug therapy (see Rapidly progressive glomerulonephritis treatment).
Third, blood purification therapy
When associated with acute renal failure or chronic renal failure have been formed by the need for hemodialysis or peritoneal dialysis.
KIDNEYSERVICE@HOTMAIL.COM
Hematuria is a dangerous signal
At a gathering of friends, W lady called me to her side. Anxious expression on her face, I infer that she must have something on his mind. Sure enough, W lady told me that when the recent discovery of two days urine wash the meat watery urine, she did not know whether the situation is serious, or need to the hospital to be checked.
After listening to Ms. W's narrative, I asked her recently whether the medication, as well as eating the food situation. Because some drugs, such as rifampicin, Dilantin, phenothiazines, as well as certain foods, such as beets, senna, etc. make urine was red peppers, looks like gross hematuria, but the urine OK routine inspection, there is no red blood cells in the urine. If red blood cells in urine routine examination under the microscope more than three numbers, then it is called hematuria medicine. In rare cases, hematuria can, after menstruation or catheter inserted pollution caused by strenuous exercise. If you have not talked about the above mentioned situations, the urine is a dangerous signal.
Some hematuria patients feel good, no discomfort, very mild hematuria or flickering, this is the patient often careless, not timely medical treatment, resulting in delayed diagnosis and treatment. According to a group of 246 cases without symptoms hematuria statistics, found that there is a serious urinary tract lesions (22%), which accounted for 10% of malignant tumors; another group of 200 cases of hematuria, and urinary tract diseases accounted for 20% of severe, malignant tumor as much as 13%. So hematuria urinary tract disorders may have serious alarm, every patient hematuria patients should conduct a detailed and systematic checks.
Young, middle-aged, elderly patients cause hematuria are not identical. Order incidence of hematuria adolescents with urinary tract infections, glomerulonephritis, is a common congenital urinary tract malformations. Middle-aged places urinary tract infections, bladder stones and tumors as much. 40 to 60 year-old patient, men with bladder cancer, kidney or ureter cancer, women with urinary tract infections, stones, bladder cancer is common. Patients over 60 years of age, men with an enlarged prostate, prostate cancer, urinary tract infections, women with bladder cancer, urinary tract infection is common.
As Ms. W, what is the reason caused hematuria, you need to carry out a series of checks to clear. General hospital examination procedure is to first define what hematuria from the site, do phase microscopy, see urine RBC morphology. Such as red blood cell morphology exotic, varied, significant differences in shape and size, the number of red blood cells per milliliter of urine over 8000, compared with glomerular disease caused by renal biopsy should be done, there is no need to do CT, cystoscopy, renal vascular there are both expensive check imaging and other damage, if the urine of normal red blood cell morphology, intravenous urography should check CT, B ultrasound, cystoscopy and so on. Through these inspections, the vast majority of patients with hematuria can determine what kind of illness due.
The next day, over the age of 50, according to my request, Ms. W was admitted to the hospital. Soon, was diagnosed with bladder cancer. Fortunately still early, surgeons carefully removed her tumor, is still alive and healthy.
After listening to Ms. W's narrative, I asked her recently whether the medication, as well as eating the food situation. Because some drugs, such as rifampicin, Dilantin, phenothiazines, as well as certain foods, such as beets, senna, etc. make urine was red peppers, looks like gross hematuria, but the urine OK routine inspection, there is no red blood cells in the urine. If red blood cells in urine routine examination under the microscope more than three numbers, then it is called hematuria medicine. In rare cases, hematuria can, after menstruation or catheter inserted pollution caused by strenuous exercise. If you have not talked about the above mentioned situations, the urine is a dangerous signal.
Some hematuria patients feel good, no discomfort, very mild hematuria or flickering, this is the patient often careless, not timely medical treatment, resulting in delayed diagnosis and treatment. According to a group of 246 cases without symptoms hematuria statistics, found that there is a serious urinary tract lesions (22%), which accounted for 10% of malignant tumors; another group of 200 cases of hematuria, and urinary tract diseases accounted for 20% of severe, malignant tumor as much as 13%. So hematuria urinary tract disorders may have serious alarm, every patient hematuria patients should conduct a detailed and systematic checks.
Young, middle-aged, elderly patients cause hematuria are not identical. Order incidence of hematuria adolescents with urinary tract infections, glomerulonephritis, is a common congenital urinary tract malformations. Middle-aged places urinary tract infections, bladder stones and tumors as much. 40 to 60 year-old patient, men with bladder cancer, kidney or ureter cancer, women with urinary tract infections, stones, bladder cancer is common. Patients over 60 years of age, men with an enlarged prostate, prostate cancer, urinary tract infections, women with bladder cancer, urinary tract infection is common.
As Ms. W, what is the reason caused hematuria, you need to carry out a series of checks to clear. General hospital examination procedure is to first define what hematuria from the site, do phase microscopy, see urine RBC morphology. Such as red blood cell morphology exotic, varied, significant differences in shape and size, the number of red blood cells per milliliter of urine over 8000, compared with glomerular disease caused by renal biopsy should be done, there is no need to do CT, cystoscopy, renal vascular there are both expensive check imaging and other damage, if the urine of normal red blood cell morphology, intravenous urography should check CT, B ultrasound, cystoscopy and so on. Through these inspections, the vast majority of patients with hematuria can determine what kind of illness due.
The next day, over the age of 50, according to my request, Ms. W was admitted to the hospital. Soon, was diagnosed with bladder cancer. Fortunately still early, surgeons carefully removed her tumor, is still alive and healthy.
Red urine is not necessarily hematuria
Red urine, microscopic examination was normal. This is not true of
hematuria.
Beets, red peppers and other certain foods, red food dye can also cause urine red; some drugs and their metabolites such as senna, rifampicin, Dilantin can also cause urine was red. In addition, intravascular hemolysis hemoglobinuria, muscle cell damage and other is red, these are not real hematuria, urine examination can identify the presence of red blood cells.
So, if there is red blood cells in urine hematuria really is it?
No. Women's menstrual period may cause pollution hematuria, urethral catheterization injury, can cause damage hematuria, in addition, some counterfeiters deliberately mixed blood in the urine can cause hematuria forgery, these cases can not be regarded as a true hematuria, urine specimen collection time, keep an eye can be excluded.
The exclusion of the above situation, urinalysis erythrocytes ≥3 / high power field or ≥8000 cells / ml, or 12 hours Urine red blood cell count more than 500,000, can be diagnosed as true hematuria. Generally believed that the emergence of true hematuria means kidney, ureter, bladder, prostate and urinary tract lesions or systemic diseases involving the urinary system to other systems due attention should be paid.
Whether it makes sense to do a true hematuria?
No. A transient hematuria, by pollen, chemicals or drugs allergy. After strenuous exercise, can occur a transient hematuria, or even gross hematuria, viral infections (such as colds) can also occur occasionally, usually without significance. Only repeatedly checked for authenticity before hematuria should pay attention to, should be further examination.
Beets, red peppers and other certain foods, red food dye can also cause urine red; some drugs and their metabolites such as senna, rifampicin, Dilantin can also cause urine was red. In addition, intravascular hemolysis hemoglobinuria, muscle cell damage and other is red, these are not real hematuria, urine examination can identify the presence of red blood cells.
So, if there is red blood cells in urine hematuria really is it?
No. Women's menstrual period may cause pollution hematuria, urethral catheterization injury, can cause damage hematuria, in addition, some counterfeiters deliberately mixed blood in the urine can cause hematuria forgery, these cases can not be regarded as a true hematuria, urine specimen collection time, keep an eye can be excluded.
The exclusion of the above situation, urinalysis erythrocytes ≥3 / high power field or ≥8000 cells / ml, or 12 hours Urine red blood cell count more than 500,000, can be diagnosed as true hematuria. Generally believed that the emergence of true hematuria means kidney, ureter, bladder, prostate and urinary tract lesions or systemic diseases involving the urinary system to other systems due attention should be paid.
Whether it makes sense to do a true hematuria?
No. A transient hematuria, by pollen, chemicals or drugs allergy. After strenuous exercise, can occur a transient hematuria, or even gross hematuria, viral infections (such as colds) can also occur occasionally, usually without significance. Only repeatedly checked for authenticity before hematuria should pay attention to, should be further examination.
Patients with systemic lupus erythematosus which performance
Systemic lupus erythematosus is a multi-system disease damage, presenting
symptoms are varied, some patients with a basic organ damage based, and
performance systems for some of the symptoms, the patient again some other organ
systems damage to the main, it is also manifested as symptoms of another system,
sometimes many organs can be damaged at the same time, the condition is more
complex, so for patients with systemic lupus erythematosus in terms of
performance is not a fixed pattern. To sum up can be divided into renal
manifestations and kidney damage performance.
(1) the performance of systemic lupus erythematosus was the diversity beyond the kidney can no obvious symptoms, and test results only erythematosus cells or positive antinuclear antibodies until fulminant dangerous. But in general the patient on medical history and clinical manifestations, more damage with multiple joint pain, fever, rash and kidney, lung, cardiovascular and blood system. The main signs and symptoms include:
① patients with fever, hot-type variable, can be divided into long-term unexplained fever, also for the heat.
②60% of patients may present both cheeks and bridge of the nose like a butterfly-shaped rash, erythema subsided after generally do not leave scars and pigmentation. The rash erythema often encountered after the sun or ultraviolet light irradiation increased, this is called light sensitive. 50% of patients seen hair loss. Also visible urticaria, erythema discoid body, around the nail erythema purple, purple haemorrhage, oral and nasal ulcers.
③90% of patients with joint pain, common in the small joints of the limbs, were multiple. About 10% of patients may have mild joint deformity.
④ pericarditis patients with active systemic lupus erythematosus may occur up to 60%, usually for brief mild clinical manifestations. 10% of patients may have myocarditis performance.
⑤40% -46% of patients with pleurisy can occur, such as chest tightness, wheezing.
⑥50% -75% of patients may have anemia; leukopenia 60% of patients, particularly lymphocytes decreased; platelets can be reduced.
⑦ some patients may have to demonstrate their nervous system. However, its performance is complex and diverse, usually mental disorders such as depression, psychosis, etc., other visible epilepsy, migraine, paralysis, disease, peripheral neuropathy and retinopathy.
⑧ other visible irregular menstruation, premenstrual symptoms, especially migraine. Some patients may occur painless lymph nodes, parotid gland enlargement.
(2) Approximately 70% of patients with varying degrees of renal impairment clinical manifestations, insidious onset may also be abrupt, the course is generally longer, with or without symptoms. Urinalysis is not normal, often hematuria and (or) proteinuria. Edema and hypertension, is also one of the common performance.
Late lupus nephritis urea disease can occur, when the clinical manifestations of lupus activity may disappear or not typical.
If you have any questions you can consult online Shijiazhuang kidney hospital doctor.kidneyservice@hotmail.com
(1) the performance of systemic lupus erythematosus was the diversity beyond the kidney can no obvious symptoms, and test results only erythematosus cells or positive antinuclear antibodies until fulminant dangerous. But in general the patient on medical history and clinical manifestations, more damage with multiple joint pain, fever, rash and kidney, lung, cardiovascular and blood system. The main signs and symptoms include:
① patients with fever, hot-type variable, can be divided into long-term unexplained fever, also for the heat.
②60% of patients may present both cheeks and bridge of the nose like a butterfly-shaped rash, erythema subsided after generally do not leave scars and pigmentation. The rash erythema often encountered after the sun or ultraviolet light irradiation increased, this is called light sensitive. 50% of patients seen hair loss. Also visible urticaria, erythema discoid body, around the nail erythema purple, purple haemorrhage, oral and nasal ulcers.
③90% of patients with joint pain, common in the small joints of the limbs, were multiple. About 10% of patients may have mild joint deformity.
④ pericarditis patients with active systemic lupus erythematosus may occur up to 60%, usually for brief mild clinical manifestations. 10% of patients may have myocarditis performance.
⑤40% -46% of patients with pleurisy can occur, such as chest tightness, wheezing.
⑥50% -75% of patients may have anemia; leukopenia 60% of patients, particularly lymphocytes decreased; platelets can be reduced.
⑦ some patients may have to demonstrate their nervous system. However, its performance is complex and diverse, usually mental disorders such as depression, psychosis, etc., other visible epilepsy, migraine, paralysis, disease, peripheral neuropathy and retinopathy.
⑧ other visible irregular menstruation, premenstrual symptoms, especially migraine. Some patients may occur painless lymph nodes, parotid gland enlargement.
(2) Approximately 70% of patients with varying degrees of renal impairment clinical manifestations, insidious onset may also be abrupt, the course is generally longer, with or without symptoms. Urinalysis is not normal, often hematuria and (or) proteinuria. Edema and hypertension, is also one of the common performance.
Late lupus nephritis urea disease can occur, when the clinical manifestations of lupus activity may disappear or not typical.
If you have any questions you can consult online Shijiazhuang kidney hospital doctor.kidneyservice@hotmail.com
2015年1月14日星期三
Is how the formation of renal cysts
Kidney cysts can be cured? Some patients lack this knowledge, I do not know
that they have with the kidney cysts, leading to disease progression.
It is not clear the reasons for its formation, said the cause of congenital and acquired causes of said support has a certain basis. About kidney cyst is how it formed? One theory is that it may be the cause of polycystic kidney disease is similar, but somewhat different in degree, but now more popular cause of polycystic kidney disease is a congenital developmental defects that it says is a congenital kidney cyst developmental defects caused by the disease.
Explore the formation of renal cysts can be found congenital and acquired etiology have some support basis and reason. In animal experiments, when the cause of renal tubular obstruction and experimental samples ischemia, can make it simple renal cysts suffer from this phenomenon suggesting that renal cysts may be acquired diseases. And there is also evidence of renal cyst formation is due to a congenital kidney defect.
Renal cysts is the general term of varying sizes that do not communicate with the outside world appeared cystic mass in the kidney, is a progressive disease, over time, gradually increasing the extension of the cyst, when the cyst near or over 3 cm to active treatment, control cyst increases, otherwise the cyst continues to increase not only the kidney capsule by pulling down the waist causing discomfort, and normal kidney cyst oppression structure, resulting in renal ischemia and hypoxia, leading to kidney fibrosis, abnormal renal function appear.
Shijiazhang Kidney Disease Hospital in China
It is not clear the reasons for its formation, said the cause of congenital and acquired causes of said support has a certain basis. About kidney cyst is how it formed? One theory is that it may be the cause of polycystic kidney disease is similar, but somewhat different in degree, but now more popular cause of polycystic kidney disease is a congenital developmental defects that it says is a congenital kidney cyst developmental defects caused by the disease.
Explore the formation of renal cysts can be found congenital and acquired etiology have some support basis and reason. In animal experiments, when the cause of renal tubular obstruction and experimental samples ischemia, can make it simple renal cysts suffer from this phenomenon suggesting that renal cysts may be acquired diseases. And there is also evidence of renal cyst formation is due to a congenital kidney defect.
Renal cysts is the general term of varying sizes that do not communicate with the outside world appeared cystic mass in the kidney, is a progressive disease, over time, gradually increasing the extension of the cyst, when the cyst near or over 3 cm to active treatment, control cyst increases, otherwise the cyst continues to increase not only the kidney capsule by pulling down the waist causing discomfort, and normal kidney cyst oppression structure, resulting in renal ischemia and hypoxia, leading to kidney fibrosis, abnormal renal function appear.
Shijiazhang Kidney Disease Hospital in China
Renal cysts can lead to kidney dysfunction
if you have any questions, you can ask Shijiazhuang Kidney Disease hospital doctor, we will give you the free help.
What are the symptoms of kidney cyst performance? Kidney cyst is a serious threat to kidney function disease, if not treated, it can cause more severe in patients with renal illness, especially those who missed the best time for treatment of patients more caution.
1, one of the common symptoms of kidney cyst is fixed to one or both of the pain, dull, and will radiate to the lower back and waist. The symptoms of kidney patients mainly because of pent traction caused water to fall.
2, renal cysts in patients with high blood pressure can also cause symptoms. Mainly due to the oppression of kidney cysts caused by ischemia, thereby enabling an increase in renin secretion due.
3, hematuria cyst symptoms caused mainly when patients with renal cysts disease onset, increased pressure causes kidney infection or blood vessel wall caused by excessive stretching and rupture occurs.
4, renal cysts can also cause kidney dysfunction, which is mainly due to cystic mass, oppression makes normal kidney tissue was significantly reduced, and eventually loss of kidney function.
5, renal cysts can also cause kidney enlargement, and this is a common symptom of renal cysts in a little more serious. Normally, the kidneys more swollen badly, the more kidney function is poor.
What are the symptoms of kidney cyst performance? Kidney cyst is a serious threat to kidney function disease, if not treated, it can cause more severe in patients with renal illness, especially those who missed the best time for treatment of patients more caution.
1, one of the common symptoms of kidney cyst is fixed to one or both of the pain, dull, and will radiate to the lower back and waist. The symptoms of kidney patients mainly because of pent traction caused water to fall.
2, renal cysts in patients with high blood pressure can also cause symptoms. Mainly due to the oppression of kidney cysts caused by ischemia, thereby enabling an increase in renin secretion due.
3, hematuria cyst symptoms caused mainly when patients with renal cysts disease onset, increased pressure causes kidney infection or blood vessel wall caused by excessive stretching and rupture occurs.
4, renal cysts can also cause kidney dysfunction, which is mainly due to cystic mass, oppression makes normal kidney tissue was significantly reduced, and eventually loss of kidney function.
5, renal cysts can also cause kidney enlargement, and this is a common symptom of renal cysts in a little more serious. Normally, the kidneys more swollen badly, the more kidney function is poor.
How do suffer from kidney cyst
Left kidney cyst is one of the more common diseases, for patients who are
left renal cyst wondering how it is, do not know why this happens, they usually
are not aware of what, why do you have this disease ?
Left kidney cyst is how is it? Experts tell us nephropathy in renal cysts are relatively neglected disease, many people feel small cysts, ignores the treatment, not knowing that it will lead to an immeasurable loss, left kidney cyst kidney cyst on the left kidney of all the symptoms collectively, in general, we believe that the left kidney cysts finger of the left renal solitary renal cysts. But there is little need to understand is that the etiology of left renal cysts but not because of congenital acquired due to pathogenic factors.
Numerous clinical and research studies guess, probably evolved by renal tubular diverticulum. Because of age, distal renal tubular diverticulum continue to increase, renal tubular cells and visceral parietal cells do not grow together to form a gap in the middle, the epithelial cells are constantly cyst fluid secretion, the gap expanded to form cysts.
What are the causes of renal cysts? We want to know the left kidney cysts in very severe cases of ruptured cyst may occur suddenly, cause kidney infections and thus make severely deteriorated condition. Due to the long-term oppression kidney cyst tissues and organs, on renal function normal operation also had a very big impact.
Left kidney cyst is how is it? Experts tell us nephropathy in renal cysts are relatively neglected disease, many people feel small cysts, ignores the treatment, not knowing that it will lead to an immeasurable loss, left kidney cyst kidney cyst on the left kidney of all the symptoms collectively, in general, we believe that the left kidney cysts finger of the left renal solitary renal cysts. But there is little need to understand is that the etiology of left renal cysts but not because of congenital acquired due to pathogenic factors.
Numerous clinical and research studies guess, probably evolved by renal tubular diverticulum. Because of age, distal renal tubular diverticulum continue to increase, renal tubular cells and visceral parietal cells do not grow together to form a gap in the middle, the epithelial cells are constantly cyst fluid secretion, the gap expanded to form cysts.
What are the causes of renal cysts? We want to know the left kidney cysts in very severe cases of ruptured cyst may occur suddenly, cause kidney infections and thus make severely deteriorated condition. Due to the long-term oppression kidney cyst tissues and organs, on renal function normal operation also had a very big impact.
2015年1月11日星期日
Can gross hematuria with clots occur in renal parenchymal disease(GN,TIN)?What renal parenchymal diseases give a combination of gross hemative and rapidly progressing renal impairment?
Can gross hematuria with clots occur in renal parenchymal disease(GN,TIN)?What renal parenchymal diseases give a combination of gross hemative and rapidly progressing renal impairment?
I have a 25 year old male patient who presented with right flank pain and gross hematuria(with clots) of a week's duration. No systemic symptoms. Urine shows RBC's and1+ proteinuria but no RBC casts. His creatinine has progressively increased from 1.6 to 4.4 mg/dl..Left kidney is small while the right is rather big(13cm) with no stones or features of obstruction. CystoUreteroscopy showed that the blood was coming out of the right ureteric orifice but the bladder and the right ureter(up until the proximal ureter ) looked normal. I have not been able to explain the rapid rise in creatinine in terms of renal hypoperfusion(with prerenal azotemia or ATN) or obstruction.He had received a contrast agent for CT and Contrast nephropathy is a possibility. The patient is clinically stable and the gross hematuria is clearing. My questions are can hematuria with clots occur in glomerular disease? Shall I just observe and see if the renal function will improve or start doing additional tests(e.g serologic tests like ANA,ANCA,etc)
Yes, gross hematuria sufficient to induce some clotting in the urine has been described rarely in glomerular disease, especially IgA nephropathy and/or in combination with a coagulopathy. Sufficient hematuria to induce clot formation can be seen with any of the crescentic GNs (ANCA, aGBM, SLE,) but is most common in IgA nephropathy. Other causes include genetic diseases like Alport’s or thin basement membrane disease. The inequality in kidney size and elevated creatinine here indicate there is bilateral renal injury, perhaps from contrast. The unilateral bleeding, and absence of proteinuria and casts would put GN low on the list of likely causes of the hematuria. It would seem more likely that something acute happened on the right – a non-opaque stone, tumor, embolus, trauma etc. With the history of recent contrast and now resolution (I assume GFR is improving as well), I do not think screening for GN is likely to be positive or alter management. I would suggest screening for coagulation disorders and obtaining optimal imaging studies of the R kidney and perhaps an angiogram to exclude any treatable entities that might represent the source of bleeding.
I have a 25 year old male patient who presented with right flank pain and gross hematuria(with clots) of a week's duration. No systemic symptoms. Urine shows RBC's and1+ proteinuria but no RBC casts. His creatinine has progressively increased from 1.6 to 4.4 mg/dl..Left kidney is small while the right is rather big(13cm) with no stones or features of obstruction. CystoUreteroscopy showed that the blood was coming out of the right ureteric orifice but the bladder and the right ureter(up until the proximal ureter ) looked normal. I have not been able to explain the rapid rise in creatinine in terms of renal hypoperfusion(with prerenal azotemia or ATN) or obstruction.He had received a contrast agent for CT and Contrast nephropathy is a possibility. The patient is clinically stable and the gross hematuria is clearing. My questions are can hematuria with clots occur in glomerular disease? Shall I just observe and see if the renal function will improve or start doing additional tests(e.g serologic tests like ANA,ANCA,etc)
Yes, gross hematuria sufficient to induce some clotting in the urine has been described rarely in glomerular disease, especially IgA nephropathy and/or in combination with a coagulopathy. Sufficient hematuria to induce clot formation can be seen with any of the crescentic GNs (ANCA, aGBM, SLE,) but is most common in IgA nephropathy. Other causes include genetic diseases like Alport’s or thin basement membrane disease. The inequality in kidney size and elevated creatinine here indicate there is bilateral renal injury, perhaps from contrast. The unilateral bleeding, and absence of proteinuria and casts would put GN low on the list of likely causes of the hematuria. It would seem more likely that something acute happened on the right – a non-opaque stone, tumor, embolus, trauma etc. With the history of recent contrast and now resolution (I assume GFR is improving as well), I do not think screening for GN is likely to be positive or alter management. I would suggest screening for coagulation disorders and obtaining optimal imaging studies of the R kidney and perhaps an angiogram to exclude any treatable entities that might represent the source of bleeding.
B-is diffuse parenchymal renal disease, renal function
Question: Hello, I took B-is diffuse parenchymal renal disease, renal
function, urine do it for three days, the first day all the normal, the next two
occult, occult third one, other indicators of normal , kidney stones, a month
ago I was in the right ureter, after extracorporeal lithotripsy left kidney
began to hurt, it hurts the right, but not as obvious to the left, according to
B-say right ureter stone row successfully, but there are still about Shen area
stones, how does that matter? Serious?
Expert answer: The volume of about 8 mm diameter slightly larger stones, but no good if renal obstruction of the urinary system, you can use gravel extracorporeal shock wave lithotripsy machine, but also by a number of endoscopy (such as kidney, ureteroscopy etc.) to take the stone or gravel. In the treatment process, but also need to closely observe the situation stone fragments discharged. Once unforeseen circumstances, you should get in touch with a doctor promptly.
Large stone volume (more than 10 mm in diameter, while hematuria, infection, tumors or urinary tract obstruction and other complications that may affect renal function should be treated with surgery, promptly remove the stones.
Expert answer: The volume of about 8 mm diameter slightly larger stones, but no good if renal obstruction of the urinary system, you can use gravel extracorporeal shock wave lithotripsy machine, but also by a number of endoscopy (such as kidney, ureteroscopy etc.) to take the stone or gravel. In the treatment process, but also need to closely observe the situation stone fragments discharged. Once unforeseen circumstances, you should get in touch with a doctor promptly.
Large stone volume (more than 10 mm in diameter, while hematuria, infection, tumors or urinary tract obstruction and other complications that may affect renal function should be treated with surgery, promptly remove the stones.
Renal hypertension and renal pathology of renal damage
Renal Parenchymal Disease: Cause, Symptoms, Treatment
May occur in patients with malignant hypertension renal artery disease small renal glomerular lesions and tubulointerstitial lesions.1, the main pathological changes in small renal artery lesions, including vascular endothelial damage and small artery involvement performance. The naked eye can see, there is no basis in patients with kidney disease, kidney size is often normal, visible under the sebum and film spotting congestion and renal medulla. Light microscopy showed small proliferative endarteritis, necrosis of small arteries and mucin-like changes in the small arteries of the typical performance is fibrinoid necrosis, intimal lipid deposition and transparent thrombosis. Vessel wall and surrounding neutrophil and mononuclear cell infiltration. Interlobular artery intima may have fibrin deposition, intimal hyperplasia associated with severe collagen fibers were concentric deposition, showing typical onion skin-like performance. Severe stenosis of the blood vessel disease that leads to complete occlusion of the vessel lumen and renal ischemia. Kidney arcuate arteries often no obvious lesions, or only in chronic hypertension.
2, the glomerular capillary loops glomerular lesions can occur periodically fibrinoid necrosis, capillary lumen thrombosis. Glomerular basement membrane and glomerular sclerosis ischemic shrinkage, may have focal mesangial cell proliferation, occasionally forming a crescent; renal cyst wall thickening.
3, tubulointerstitial renal tubular epithelial cell loss can occur, such as acute tubular necrosis regeneration lesions; may have varying degrees of renal tubular atrophy. Visible renal interstitial edema, inflammatory cell infiltration can. As the disease progresses, there may be different degrees of fibrosis.
4, primary malignant hypertension and renal parenchymal renal pathology at different points renal artery had severe damage is a common feature of the two, but because of the pathogenesis of different diseases, there are two different pathological features.
Renal artery disease: primary malignant hypertension small artery disease seen in the acute phase of endothelial cell degeneration, loss and endometrial edema, severe and visible intimal wall fibrinoid necrosis, thrombosis. Chronic phase endometrium was onion skin-like fibrous thickening, stenosis. Renal artery mainly small wall thickening, small artery hyaline degeneration.
Glomerular lesions: Primary malignant hypertension glomerular lesions mainly glomerular basement membrane (GBM) and ischemic ischemic shrinkage sclerosis, severe cases can occur periodically fibrinoid necrosis. Renal glomerular inflammatory lesions mainly glomerular injury, mesangial matrix increase or glomerular sclerosis, sometimes visible typical variety of glomerular pathology.
Between tubular and renal interstitial disease: acute renal tubular injury with diffuse primary malignant hypertension, and degeneration, atrophy and regeneration, renal interstitial edema, sometimes showing lymphoid and monocyte infiltration spotty. Compared with renal parenchymal renal tubules and interstitial chronic injury, namely tubular atrophy and spotty compensatory hypertrophy, renal interstitial multifocal lymphoid and mononuclear cell infiltration and fibrosis.
2015年1月10日星期六
With plasmaphérès treat chronic renal failure in stage 4 attracted chronic glomerulonephritis
1.The medical record summary
1. the patient Li Xiaoxia, 43, comes from Huxian district of the province of Xi'an
2. the symptoms described by the patient: intermittent edema of the lower extremities for five years, fatigue, abnormal renal function 3 months.
3. history of the disease: before age 5, he has no obvious incentive, the patient appeared edema of the lower extremities, no rash, no joint pain, went to the doctor at a local hospital to check the urine, examination showed: protein 2, occult blood +, she took Chinese edicinales herbesm in 2 years, and it has shown no significant improvement in the review, and then she is went to the doctor at the hospital accessory Jiaotong University in Xi'an, in June 2009, the B-ultrasound shows: 9.5cm the left kidney, right kidney 9.1 cm; Renal biopsy showed: chronic renal failure attracted chronic glomerulonephritis, give prednisone 40 mg orally, take it every morning in 2 months, still no significant improvement; receive treatment in a hospital in Xi'an nephropathy in August 2009, gives antipyretic tablet orally nephritis, adheres to a one-month treatment, the urine test had no significant change continues processing taking Chinese medicines; there three months she felt lfaible, and tests of renal function showed: blood creatinine 291.5umol / L, uric acid 599umol / L, hemoglobin 67g / L For more effective treatment, the patient arrives at our hospital.
4.temps inlet
On February 19, 2012
2..Les admission tests:
1. Arterial voltage: 130 / 90mmHg, 164cm height, weight 47 kg
2. urine test for protein + 2, + occult blood
3. The proportion of the morning urine: 1.008, pH 5 5, urine RBC52.40 / ul fine granule tubes 25-30 / HP
4. urinary excretion of protein 24 hours: 2.59 g / 24h
5.Sang: total protein 58.6g / L Albumin 35.4g / L ALT5.1U / L, AST 11.4U / L, hemoglobin 92g L
6 .Analyse blood lipids: TG 2.18mmol / L CHOL3.48mmol / L HDL 0.75mmol / L LDL1.74mmol / L Lpa129mg / L
Blood creatinine 317umol / L urea nitrogen 12.4mmol / L, uric acid 501umol / l
Blood: K + 4.08mmol / L, Na + 142mmol / L, Cl-108mmol / L Ca1.8 mmol / L, CO2 16.7mmol / L PO4 1.89mmol / L
3.La admission diagnosis
Initial disease diagnosis: chronic glomerulonephritis, nephropathy AIGA
Functional diagnosis: chronic renal failure in stage 4
The diagnosis of complications: renal hypertension, renal anemia, electrolyte imbalance and imbalance of acid-base.
4.l'effet treatment
1. After admission, treat complications with Western medicine; the exchange of plasma treatment was given to 2 March
2 Micro-Chinese medicine, prevent renal fibrosis and to prevent the reaction of the ignition of an injury;
3, repair the results of treatment were as shown --Bra:
plasma
Arterial tension draft proportion globule protein
Before treatment 130/90 1008 2 52.40 / ul
After treatment 100/70 1.025 + 8.6 / ul
urinary protein 24 hour tube k + Na + Cl- Co2 PO4
2.59 25-30 4.08 142 108 16.7 1.89
1.23 8-10 4.3 137 102 22 0.93
Albumin creatinine blood urea nitrogen uric acid
35.4g / L 12.4 317 501
45.6g / L 257 336 9.6
5.The views of experts:
1.la this patient nephropathy is caused by immune injury
2.la patient reaches the immune injury for these reasons:
①D'abord, many of IgA to start the inflammatory response
② damage the function of glomerular and tubular function;
③ started renal fibrosis, fibrosis has not been developed a controlled portion of the scar tissue, promotes the deterioration of renal function
3. cold bactériennese of infections such as by the antigen-mediated immune response in patients with glomerular damage intrinsic Immunotherapy.
4, the effect of plasmapheresis treatment is to clear the objects of immune injury in the patient, to reduce kidney damage; and clean in the disordered state of the environment in and promote a stable arterial pressure and do normal endocrine kidney.
5, the cleaning of a large number of immune damage substances, plasmapheresis clean obstacles for the recovery of the kidneys and activate the function of Immunotherapy, in inhibiting the inflammatory response and to improve the local microcirculation.
1. the patient Li Xiaoxia, 43, comes from Huxian district of the province of Xi'an
2. the symptoms described by the patient: intermittent edema of the lower extremities for five years, fatigue, abnormal renal function 3 months.
3. history of the disease: before age 5, he has no obvious incentive, the patient appeared edema of the lower extremities, no rash, no joint pain, went to the doctor at a local hospital to check the urine, examination showed: protein 2, occult blood +, she took Chinese edicinales herbesm in 2 years, and it has shown no significant improvement in the review, and then she is went to the doctor at the hospital accessory Jiaotong University in Xi'an, in June 2009, the B-ultrasound shows: 9.5cm the left kidney, right kidney 9.1 cm; Renal biopsy showed: chronic renal failure attracted chronic glomerulonephritis, give prednisone 40 mg orally, take it every morning in 2 months, still no significant improvement; receive treatment in a hospital in Xi'an nephropathy in August 2009, gives antipyretic tablet orally nephritis, adheres to a one-month treatment, the urine test had no significant change continues processing taking Chinese medicines; there three months she felt lfaible, and tests of renal function showed: blood creatinine 291.5umol / L, uric acid 599umol / L, hemoglobin 67g / L For more effective treatment, the patient arrives at our hospital.
4.temps inlet
On February 19, 2012
2..Les admission tests:
1. Arterial voltage: 130 / 90mmHg, 164cm height, weight 47 kg
2. urine test for protein + 2, + occult blood
3. The proportion of the morning urine: 1.008, pH 5 5, urine RBC52.40 / ul fine granule tubes 25-30 / HP
4. urinary excretion of protein 24 hours: 2.59 g / 24h
5.Sang: total protein 58.6g / L Albumin 35.4g / L ALT5.1U / L, AST 11.4U / L, hemoglobin 92g L
6 .Analyse blood lipids: TG 2.18mmol / L CHOL3.48mmol / L HDL 0.75mmol / L LDL1.74mmol / L Lpa129mg / L
Blood creatinine 317umol / L urea nitrogen 12.4mmol / L, uric acid 501umol / l
Blood: K + 4.08mmol / L, Na + 142mmol / L, Cl-108mmol / L Ca1.8 mmol / L, CO2 16.7mmol / L PO4 1.89mmol / L
3.La admission diagnosis
Initial disease diagnosis: chronic glomerulonephritis, nephropathy AIGA
Functional diagnosis: chronic renal failure in stage 4
The diagnosis of complications: renal hypertension, renal anemia, electrolyte imbalance and imbalance of acid-base.
4.l'effet treatment
1. After admission, treat complications with Western medicine; the exchange of plasma treatment was given to 2 March
2 Micro-Chinese medicine, prevent renal fibrosis and to prevent the reaction of the ignition of an injury;
3, repair the results of treatment were as shown --Bra:
plasma
Arterial tension draft proportion globule protein
Before treatment 130/90 1008 2 52.40 / ul
After treatment 100/70 1.025 + 8.6 / ul
urinary protein 24 hour tube k + Na + Cl- Co2 PO4
2.59 25-30 4.08 142 108 16.7 1.89
1.23 8-10 4.3 137 102 22 0.93
Albumin creatinine blood urea nitrogen uric acid
35.4g / L 12.4 317 501
45.6g / L 257 336 9.6
5.The views of experts:
1.la this patient nephropathy is caused by immune injury
2.la patient reaches the immune injury for these reasons:
①D'abord, many of IgA to start the inflammatory response
② damage the function of glomerular and tubular function;
③ started renal fibrosis, fibrosis has not been developed a controlled portion of the scar tissue, promotes the deterioration of renal function
3. cold bactériennese of infections such as by the antigen-mediated immune response in patients with glomerular damage intrinsic Immunotherapy.
4, the effect of plasmapheresis treatment is to clear the objects of immune injury in the patient, to reduce kidney damage; and clean in the disordered state of the environment in and promote a stable arterial pressure and do normal endocrine kidney.
5, the cleaning of a large number of immune damage substances, plasmapheresis clean obstacles for the recovery of the kidneys and activate the function of Immunotherapy, in inhibiting the inflammatory response and to improve the local microcirculation.
2015年1月5日星期一
Eat instant noodles can lead to renal failure
Renal failure refers to kidney failure, chronic and acute points. Renal
failure patients often accompanied by high blood pressure, anemia, water and
electrolyte imbalance and other symptoms. In recent years, increased the number
of patients with renal failure, in addition to life stress factors caused by
poor eating habits is one of the incentives.
1, fullness
Overeating, excessive burden on the kidneys, kidney disease, many people are unaware that "eating out". The kidneys are important in the human body excrete waste and toxins responsible for organ. Eating too much will increase the burden on the kidneys, long-term high work load, kidneys naturally susceptible to illness.
2, salty
Eat salty also easily lead to high blood pressure. Hypertension is a systemic disease, and kidney disease often it is a pair of "twin sister." In recent years, high blood pressure and kidney damage caused by the rising incidence. Therefore, the daily life should insist salt diet, daily intake of salt should be more appropriate at 5-6 grams.
3, too sweet, too Oil
Eat too sweet, too much oil will not only increase the burden on the kidneys, but also lead to obesity. Obesity can lead to kidney fat content increases, an increase in weight, volume increases, glomerular hypertrophy. Obese patients are also prone to insulin resistance, and thus lead to diabetes. With nearly 40% of diabetic patients will be diabetic nephropathy, kidney disease, and this is one of the most difficult to treat kidney disease.
4, excessive intake of protein
Eating too much protein can cause the body's uric acid concentration, causing hyperuricemia. Elevated serum uric acid concentration in the kidney toxicity is very large, it is likely to cause renal tubular and interstitial lesions, and finally develop into chronic renal failure and uremia.
Is not appropriate to eat instant noodles, instant noodles easily lead to kidney failure
Instant noodles are a favorite of office workers, sometimes because of work pressure of time, or overtime plus late, nothing to eat, open a packet of instant noodles bubble you can eat, but should not eat too much instant noodles, the human body has a very great harm.
1, eat instant noodles could lead to kidney failure
Fried noodles are generally required because subtract fried surface water, can extend shelf life. But these oils after oxidation becomes "LPO" posted on the bread, when people eat into the body will accumulate in the blood vessels or other organs, accelerate human aging rate and cause atherosclerosis, leading to stroke, heart disease, kidney disease.
2, absolutely no nutrition are all fat
First, most of the noodles are made into too fried, bread itself contains a higher oil. In addition, to the delicious seasoning packets of instant noodles are usually 2 to 3 bags, a liquid oil, add the sauce packet or animal oil; the other is pink bag of salt, MSG, spices and other mixed; some have a small amount of dehydration package vegetables. Largest component of seasoning packet is fat. If the sauce packets, typically more than 50% fat content. If it is oil, fat contained more than 95%. The vegetables in the package because of severe dehydration vegetables basically no nutrition to speak of.
3, you stupid old eating instant noodles
This is not alarmist, the study found must maintain and enhance the normal physiological function of the brain, must be lecithin, protein, sugar, calcium, vitamin A, vitamin C, vitamin E and B vitamins and other nutrients eight. The delicious and convenient instant noodles, but totally do not have more than a variety of nutrients, the brain will often eat long-term lack of nutrition and stupid.
Since there are so many of the drawbacks of instant noodles, why are people having left it abandoned? The reason many, which is nothing less than instant noodles really convenient because you can eat right away. In fact, many healthy foods are also easy to meet your needs.
4, instant noodles too easily lead to hypertension salt content
A packet of instant noodles, the salt content is usually 2 to 3 grams, three meals a day if you eat instant noodles, daily intake of sodium content will be exceeded, the probability of suffering from high blood pressure will rise. And most people would prefer to eat instant noodles when coupled with ham, sausage sodium content is considerable. If you often eat together two match, the chances of suffering from high blood pressure will be significantly increased several times.
1, fullness
Overeating, excessive burden on the kidneys, kidney disease, many people are unaware that "eating out". The kidneys are important in the human body excrete waste and toxins responsible for organ. Eating too much will increase the burden on the kidneys, long-term high work load, kidneys naturally susceptible to illness.
2, salty
Eat salty also easily lead to high blood pressure. Hypertension is a systemic disease, and kidney disease often it is a pair of "twin sister." In recent years, high blood pressure and kidney damage caused by the rising incidence. Therefore, the daily life should insist salt diet, daily intake of salt should be more appropriate at 5-6 grams.
3, too sweet, too Oil
Eat too sweet, too much oil will not only increase the burden on the kidneys, but also lead to obesity. Obesity can lead to kidney fat content increases, an increase in weight, volume increases, glomerular hypertrophy. Obese patients are also prone to insulin resistance, and thus lead to diabetes. With nearly 40% of diabetic patients will be diabetic nephropathy, kidney disease, and this is one of the most difficult to treat kidney disease.
4, excessive intake of protein
Eating too much protein can cause the body's uric acid concentration, causing hyperuricemia. Elevated serum uric acid concentration in the kidney toxicity is very large, it is likely to cause renal tubular and interstitial lesions, and finally develop into chronic renal failure and uremia.
Is not appropriate to eat instant noodles, instant noodles easily lead to kidney failure
Instant noodles are a favorite of office workers, sometimes because of work pressure of time, or overtime plus late, nothing to eat, open a packet of instant noodles bubble you can eat, but should not eat too much instant noodles, the human body has a very great harm.
1, eat instant noodles could lead to kidney failure
Fried noodles are generally required because subtract fried surface water, can extend shelf life. But these oils after oxidation becomes "LPO" posted on the bread, when people eat into the body will accumulate in the blood vessels or other organs, accelerate human aging rate and cause atherosclerosis, leading to stroke, heart disease, kidney disease.
2, absolutely no nutrition are all fat
First, most of the noodles are made into too fried, bread itself contains a higher oil. In addition, to the delicious seasoning packets of instant noodles are usually 2 to 3 bags, a liquid oil, add the sauce packet or animal oil; the other is pink bag of salt, MSG, spices and other mixed; some have a small amount of dehydration package vegetables. Largest component of seasoning packet is fat. If the sauce packets, typically more than 50% fat content. If it is oil, fat contained more than 95%. The vegetables in the package because of severe dehydration vegetables basically no nutrition to speak of.
3, you stupid old eating instant noodles
This is not alarmist, the study found must maintain and enhance the normal physiological function of the brain, must be lecithin, protein, sugar, calcium, vitamin A, vitamin C, vitamin E and B vitamins and other nutrients eight. The delicious and convenient instant noodles, but totally do not have more than a variety of nutrients, the brain will often eat long-term lack of nutrition and stupid.
Since there are so many of the drawbacks of instant noodles, why are people having left it abandoned? The reason many, which is nothing less than instant noodles really convenient because you can eat right away. In fact, many healthy foods are also easy to meet your needs.
4, instant noodles too easily lead to hypertension salt content
A packet of instant noodles, the salt content is usually 2 to 3 grams, three meals a day if you eat instant noodles, daily intake of sodium content will be exceeded, the probability of suffering from high blood pressure will rise. And most people would prefer to eat instant noodles when coupled with ham, sausage sodium content is considerable. If you often eat together two match, the chances of suffering from high blood pressure will be significantly increased several times.
Diarrhea, acute renal failure, oliguria to be alert
Even a little diarrhea can cause acute renal failure? It turned out that the
human body can cause severe diarrhea and dehydration, if you do not replenish
moisture, renal ischemia situation will arise, causing acute renal failure. If
severe diarrhea accompanied by oliguria, or even no symptoms of the solution,
you should seek immediate medical attention.
Diarrhea did not care, and eventually even lead to kidney failure it! Xiao Zhou is a representative of the company, three days ago, and friends gathering to eat hot pot restaurants. After returning home he began to stop diarrhea, toss a night is not enough, the next morning and pulled several times. Xiao Zhou too troublesome to the hospital, they casually ate diarrhea medicine. Eat for two days of medicine, diarrhea stopped, may appear small weeks furrow eyes, his face sallow, urine solution no symptoms. Doctors quickly diagnosed, Xiao Zhou is acute renal failure caused by severe diarrhea.
Why would hurt diarrhea kidney?
? Why diarrhea will hurt the cause of acute renal failure, the kidneys it is mainly divided into three categories: pre-renal, renal parenchymal after renal. Among them, the most common pre-renal renal failure, mainly due to a serious shortage of effective blood volume leads to renal hypoperfusion blood and cause kidney dysfunction. Xiao Zhou diarrhea causes the body to dehydration, electrolyte imbalance, loss of nutrients, the body's internal environment destruction. Patients with severe water shortages will, if not able to replenish moisture, under hypovolemia case, there will be cases of renal ischemia, causing acute renal failure.
Diarrhea, acute renal failure will lead to it? Experts say not necessarily. Severe diarrhea leading to higher risk of renal failure, renal failure, low probability of mild diarrhea caused. If diarrhea, nausea, decreased amount of urine, weakness, leg edema and other symptoms, be sure to consult a doctor immediately, to exclude the possibility of acute renal failure.
Diagnosed with acute renal failure, after early treatment, most patients can resume normal renal function. Clinical data show that kidney function is still 10% to 20% of patients can not be fully restored, and gradually develop chronic renal failure, requiring long-term dialysis. So, do not diarrhea also carrying avoid catastrophe, lifelong regret.
How to prevent dehydration?
Recognize early symptoms of dehydration, especially in mild to moderate dehydration, family therapy can begin promptly. For mild dehydration, it can be a way of oral fluids. The best homemade rehydration drink soup. (Rice soup production methods: first boil a liter of water, then pour a bowl of rice, then boiled for 5 to 10 minutes, until a thin paste of water into the cooked rice into the container, add a tablespoon of sugar. and salt. when the liquid to be cooled down to room temperature, a thin paste, rice on the production well.
Secondly, you can go to the drugstore to buy oral rehydration salts, is one good way to correct dehydration. For older children, but also drinking beverages containing electrolytes and sugar sold in supermarkets. Because of these correct dehydration fluid smells bad, children do not want to accept. Should take a few times approach. If vomiting occurs after the baby is taking it from a small start, every 10 to 15 ml every 15 to 30 minutes. After the baby can tolerate, and added to each 30 ml, 60 ml. If the baby continues to diarrhea, you do not need to limit the total amount of children taking.
Critical fluids is uniform, slow. Especially in patients with small babies. Sometimes, in order to adjust the speed of the liquid to drink, the liquid can be dipped in the towel and let the baby sucking towels. More than 1 year old child can also be used to suck popsicles way. If, after the family are not satisfied after rehydration therapy, has grown to one of severe dehydration, or factors that cause dehydration persists, it should go to the hospital for a doctor's guidance, if necessary, intravenous infusion.
Diarrhea did not care, and eventually even lead to kidney failure it! Xiao Zhou is a representative of the company, three days ago, and friends gathering to eat hot pot restaurants. After returning home he began to stop diarrhea, toss a night is not enough, the next morning and pulled several times. Xiao Zhou too troublesome to the hospital, they casually ate diarrhea medicine. Eat for two days of medicine, diarrhea stopped, may appear small weeks furrow eyes, his face sallow, urine solution no symptoms. Doctors quickly diagnosed, Xiao Zhou is acute renal failure caused by severe diarrhea.
Why would hurt diarrhea kidney?
? Why diarrhea will hurt the cause of acute renal failure, the kidneys it is mainly divided into three categories: pre-renal, renal parenchymal after renal. Among them, the most common pre-renal renal failure, mainly due to a serious shortage of effective blood volume leads to renal hypoperfusion blood and cause kidney dysfunction. Xiao Zhou diarrhea causes the body to dehydration, electrolyte imbalance, loss of nutrients, the body's internal environment destruction. Patients with severe water shortages will, if not able to replenish moisture, under hypovolemia case, there will be cases of renal ischemia, causing acute renal failure.
Diarrhea, acute renal failure will lead to it? Experts say not necessarily. Severe diarrhea leading to higher risk of renal failure, renal failure, low probability of mild diarrhea caused. If diarrhea, nausea, decreased amount of urine, weakness, leg edema and other symptoms, be sure to consult a doctor immediately, to exclude the possibility of acute renal failure.
Diagnosed with acute renal failure, after early treatment, most patients can resume normal renal function. Clinical data show that kidney function is still 10% to 20% of patients can not be fully restored, and gradually develop chronic renal failure, requiring long-term dialysis. So, do not diarrhea also carrying avoid catastrophe, lifelong regret.
How to prevent dehydration?
Recognize early symptoms of dehydration, especially in mild to moderate dehydration, family therapy can begin promptly. For mild dehydration, it can be a way of oral fluids. The best homemade rehydration drink soup. (Rice soup production methods: first boil a liter of water, then pour a bowl of rice, then boiled for 5 to 10 minutes, until a thin paste of water into the cooked rice into the container, add a tablespoon of sugar. and salt. when the liquid to be cooled down to room temperature, a thin paste, rice on the production well.
Secondly, you can go to the drugstore to buy oral rehydration salts, is one good way to correct dehydration. For older children, but also drinking beverages containing electrolytes and sugar sold in supermarkets. Because of these correct dehydration fluid smells bad, children do not want to accept. Should take a few times approach. If vomiting occurs after the baby is taking it from a small start, every 10 to 15 ml every 15 to 30 minutes. After the baby can tolerate, and added to each 30 ml, 60 ml. If the baby continues to diarrhea, you do not need to limit the total amount of children taking.
Critical fluids is uniform, slow. Especially in patients with small babies. Sometimes, in order to adjust the speed of the liquid to drink, the liquid can be dipped in the towel and let the baby sucking towels. More than 1 year old child can also be used to suck popsicles way. If, after the family are not satisfied after rehydration therapy, has grown to one of severe dehydration, or factors that cause dehydration persists, it should go to the hospital for a doctor's guidance, if necessary, intravenous infusion.
2015年1月4日星期日
What is kidney failure?
Renal failure is the development of a variety of chronic kidney disease to
kidney function caused by late or part of a pathological state of all the loss.
Progression of renal failure can be divided into acute renal failure and chronic
renal failure, acute renal failure quickly, usually caused by inadequate blood
supply of the kidneys (such as trauma or burns), kidney function due to some
factors that cause damage or obstruction by poison injury, acute renal failure
caused by generation. The main reason for the long-term chronic renal failure
kidney disease, along with the time and the disease, decreased kidney function,
resulting in renal failure.
1. Acute renal failure
Rapid progression of acute renal failure, renal blood flow is usually due to insufficient supply (such as scratches or burns), renal obstruction due to some factors that cause functional impairment or hurt by poison, causing acute renal failure generation.
2. Chronic renal failure
Chronic renal failure refers to the slowly progressive renal damage caused by a variety of kidney disease eventually lead to a complete loss of uremia and renal function, causing a series of clinical symptoms and biochemical composition of endocrine metabolic disorders such as clinical syndrome from primary disease onset to begin with renal insufficiency, the interval can be several years to ten years.
Cause of disease
1. Acute renal failure
Usually caused by insufficient blood supply of the kidneys (such as scratches or burns), renal obstruction due to some factors that cause functional impairment or hurt by poison, causing acute renal failure generation.
2. Chronic renal failure
Because long-term kidney disease, along with the time and the disease, decreased kidney function, resulting in renal failure.
Clinical manifestations
1. oliguria
The most critical stage of the disease, a serious disturbance in the environment. Patients may oliguria (<400ml / day) or no urine (<100ml / day), low urine specific gravity (1.010 ~ 1.020), urinary sodium, hematuria, proteinuria, tubular urine. Water intoxication can occur in patients with severe hyperkalemia (often do this on the cause of death), metabolic acidosis (can promote hyperkalemia occurred) and azotemia (progressive increase can occur uremia), etc. endanger the lives of patients. This period of a few days to a few weeks, the longer the duration, the worse the prognosis.
2. polyuria
After increasing urine oliguria, when daily urine output of more than 500ml, enter polyuria. Since then, the daily urine output doubled, the highest daily urine 3000-6000ml, even up 10000ml more. In the initial period polyuria, urine output, although increased, but the renal clearance rate is still low, the accumulation of metabolites still exists. About 4 to 5 days, serum urea nitrogen, creatinine and decreased with increased urine output, along with improvement in symptoms of uremia. Potassium, sodium, chloride and other electrolytes in the urine may lead to a large number of discharged electrolyte imbalance or dehydration, should pay attention to the peak stage of oliguria may change as hypokalemia. This period lasts 1 to 2 weeks.
3. recovery
Urine gradually returned to normal, 3 to 12 months, the gradual recovery of renal function, renal function in most patients recovered to normal levels, only a minority of patients with chronic renal failure.
1. Acute renal failure
Rapid progression of acute renal failure, renal blood flow is usually due to insufficient supply (such as scratches or burns), renal obstruction due to some factors that cause functional impairment or hurt by poison, causing acute renal failure generation.
2. Chronic renal failure
Chronic renal failure refers to the slowly progressive renal damage caused by a variety of kidney disease eventually lead to a complete loss of uremia and renal function, causing a series of clinical symptoms and biochemical composition of endocrine metabolic disorders such as clinical syndrome from primary disease onset to begin with renal insufficiency, the interval can be several years to ten years.
Cause of disease
1. Acute renal failure
Usually caused by insufficient blood supply of the kidneys (such as scratches or burns), renal obstruction due to some factors that cause functional impairment or hurt by poison, causing acute renal failure generation.
2. Chronic renal failure
Because long-term kidney disease, along with the time and the disease, decreased kidney function, resulting in renal failure.
Clinical manifestations
1. oliguria
The most critical stage of the disease, a serious disturbance in the environment. Patients may oliguria (<400ml / day) or no urine (<100ml / day), low urine specific gravity (1.010 ~ 1.020), urinary sodium, hematuria, proteinuria, tubular urine. Water intoxication can occur in patients with severe hyperkalemia (often do this on the cause of death), metabolic acidosis (can promote hyperkalemia occurred) and azotemia (progressive increase can occur uremia), etc. endanger the lives of patients. This period of a few days to a few weeks, the longer the duration, the worse the prognosis.
2. polyuria
After increasing urine oliguria, when daily urine output of more than 500ml, enter polyuria. Since then, the daily urine output doubled, the highest daily urine 3000-6000ml, even up 10000ml more. In the initial period polyuria, urine output, although increased, but the renal clearance rate is still low, the accumulation of metabolites still exists. About 4 to 5 days, serum urea nitrogen, creatinine and decreased with increased urine output, along with improvement in symptoms of uremia. Potassium, sodium, chloride and other electrolytes in the urine may lead to a large number of discharged electrolyte imbalance or dehydration, should pay attention to the peak stage of oliguria may change as hypokalemia. This period lasts 1 to 2 weeks.
3. recovery
Urine gradually returned to normal, 3 to 12 months, the gradual recovery of renal function, renal function in most patients recovered to normal levels, only a minority of patients with chronic renal failure.
2015年1月2日星期五
Why polycystic kidney disease causes high creatinine levels in nephrotic syndrome nephrotic syndrome in Western medicine can cure it
High creatinine marked renal dysfunction, metabolic waste and toxins and
other difficult to remove creatinine from the blood. Polycystic kidney disease
is a hereditary kidney disease, characterized by a large number of renal fluid
filled cysts two kidneys. It is a chronic progressive renal disease, which will
eventually damage the kidneys and renal structure and progress to become renal
failure. Serum creatinine levels remain high, meaning more than 50% of renal
function has been impaired.
The number and size of cysts, and over time they will be oppressed and occupied space, normal kidney tissue in gradual decline and loss of renal function caused.
Early signs of kidney damage is concentrated in renal function decline. Advances in PKD patients compared to obvious clinical symptoms, such as low back pain, many patients still have normal kidney function. However, once kidney function began to decline, even a decline in glomerular filtration rate annually 4.4-5.5ml / min. It requires about 10 years of progression to end-stage renal failure, renal damage of these cysts.
Since we already know that high levels of creatinine root cause of polycystic kidney disease, we should know how to reduce radically. This is to remove these renal cysts, relieve pressure on all sides of the kidney inherent cells and tissues, kidney damage repair, improve kidney filtration function, in addition to manipulating the good, its symptoms and complications, so too will be eradicated in the blood creatinine, high creatinine will reduce natural.
PKD patients, surgical removal of large renal cysts is an immediate relief, but it can not remove all of the cysts. More importantly, because it can not solve the root of the problem, and further more cystic fluid secreted continuously down, there will be more in the kidney cyst formation.
Different surgical, CKD cell activation immunotherapy can not get rid of cysts, but affect the overall atrophy, all cyst. It not only can reduce the cysts, but also to prevent the spread of more cysts, the recurrence rate is much lower, since it is possible to suppress the source of the cyst wall of the epithelial cells, cyst fluid.
More importantly, in the micro-penetration of traditional Chinese medicine treatment of active ingredients can dilate blood vessels, promote blood circulation, relieve the number of renal ischemia, hypoxia, thus providing more blood, oxygen and nutrients needed to nourish the kidney tissue necrosis improve renal function.
The number and size of cysts, and over time they will be oppressed and occupied space, normal kidney tissue in gradual decline and loss of renal function caused.
Early signs of kidney damage is concentrated in renal function decline. Advances in PKD patients compared to obvious clinical symptoms, such as low back pain, many patients still have normal kidney function. However, once kidney function began to decline, even a decline in glomerular filtration rate annually 4.4-5.5ml / min. It requires about 10 years of progression to end-stage renal failure, renal damage of these cysts.
Since we already know that high levels of creatinine root cause of polycystic kidney disease, we should know how to reduce radically. This is to remove these renal cysts, relieve pressure on all sides of the kidney inherent cells and tissues, kidney damage repair, improve kidney filtration function, in addition to manipulating the good, its symptoms and complications, so too will be eradicated in the blood creatinine, high creatinine will reduce natural.
PKD patients, surgical removal of large renal cysts is an immediate relief, but it can not remove all of the cysts. More importantly, because it can not solve the root of the problem, and further more cystic fluid secreted continuously down, there will be more in the kidney cyst formation.
Different surgical, CKD cell activation immunotherapy can not get rid of cysts, but affect the overall atrophy, all cyst. It not only can reduce the cysts, but also to prevent the spread of more cysts, the recurrence rate is much lower, since it is possible to suppress the source of the cyst wall of the epithelial cells, cyst fluid.
More importantly, in the micro-penetration of traditional Chinese medicine treatment of active ingredients can dilate blood vessels, promote blood circulation, relieve the number of renal ischemia, hypoxia, thus providing more blood, oxygen and nutrients needed to nourish the kidney tissue necrosis improve renal function.
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