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The reason why Hypertension can happen to Patients with Chronic Renal Failure
is complex. For Chronic Renal Failure patients in early stage, the change of
exchangeable sodium in the body can not be easily detected. For Chronic Renal
Failure patients, the change of blood pressure and their sensitivity to salt are
higher than normal people.
For most Chronic Renal Failure patients in end stage, their Hypertension is
caused by water and sodium retention, or the increase of blood volume and
extracellular fluid volume. At this moment, the increased level of blood
pressure has a perfect positive correlation with salt intake. Stroke volume of
patients increases while plasma renin activity decreases. Later stroke volume
may return normal, but obstruction in peripheral vessels can be heightened. At
the moment, anemia and arteriovenous fistula also can aggravate the load of
heart. After taking dialysis, blood pressure can reduce to normal level due to
dehydration.
For a small part of patients, there may appear water volume declined and
plasma renin activity increased due to various factors such as vomiting or
diarrhea. At the moment, if patients take dialysis, there may occur obvious
raise of blood pressure due to dehydration, leading to stubborn Hypertension.
Under this situation, it is better to take several types of hypotensor at the
same time in order to control blood pressure. Some reports also say that Renal
Failure Hypertension is caused when high level inhibitor of sodium transport
directly or indirectly increase the concentration of calcium ion in smooth
muscle cells, and then lead to vasoconstriction, causing increase of blood
pressure.
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